GAP1-InsP4 BP Polyclonal Antibody, 100ul Low-speed Centrifuges Defects in CNGB1 are a

Defects in CNGB1 are a cause of cause of retinitis pigmentosa type 45

The protein can activate the complement pathway in association with MASPs and sMAP

The encoded protein binds to phosphoinositides in the plasma membrane via its C-terminal region and probably functions as a membrane-bound transcription regulator that translocates to the nucleus in response to phosphoinositide hydrolysis| for instance| induced by G-protein-coupled-receptor signaling

Caveolae contain truncated forms of this protein and may be the site of phosphorylation-dependent proteolysis

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GAP1-InsP4 BP Polyclonal Antibody, 100ul Low-speed Centrifuges Defects in CNGB1 are aRASA3 encodes a protein that binds inositol 1,3,4,5 tetrakisphosphate and stimulates the GTPase activity of Ras p21. Ras GTPase activating protein 3 functions as a negative regulator of the Ras signalling pathway. It is localized to the cell membrane via a pleckstrin homology (PH) domain in the C terminal region. Alternative splicing results in multiple transcript variants.